Professor Hsu is a Physician-Scientist in the Department of Gastroenterology and Hepatology at Taipei Veterans General Hospital. His clinical practice and translational research primarily focus on liver cirrhosis, portal hypertension, and hepatic encephalopathy. Notably, his pioneering research demonstrating that enhancing meningeal lymphangiogenesis mitigates neuroinflammation and HE—initiated during a 2018 fellowship at Yale University and published in Gastroenterology—earned him the prestigious 2023 Ta-You Wu Memorial Award. His laboratory is currently actively working to translate these findings to human patients. In addition to hepatology, Professor Hsu specializes in pancreatic cancer research, utilizing comprehensive data analysis to optimize clinical treatments that integrate medical oncology with advanced modalities like carbon ion radiation therapy.
Liver Cirrhosis, Hepatic Encephalopathy, Portal Hypertension, Pancreatic Cancer.
Pancreatic ductal adenocarcinoma (PDAC) remains one of the most lethal malignancies, historically characterized by resistance to conventional therapies and an immunosuppressive microenvironment. However, recent breakthroughs in KRAS-targeted therapies and therapeutic vaccines are rapidly transforming the treatment landscape.
For decades, KRAS—mutated in over 90% of PDAC cases—was deemed “undruggable.” The advent of novel mutation-specific (e.g., G12D, G12C) and pan-KRAS inhibitors has shattered this paradigm. Emerging agents have demonstrated profound clinical efficacy in advanced PDAC, significantly extending survival and offering a highly targeted, tolerable alternative to standard cytotoxic chemotherapy.
In tandem with targeted agents, cancer vaccines are successfully engineering robust anti-tumor immunity. An individualized mRNA neoantigen vaccine has shown the ability to induce de novo, long-lasting T-cell responses and significantly delay recurrence in the adjuvant setting. Concurrently, off-the-shelf platforms have elicited potent KRAS-specific T-cell responses in trials, effectively targeting minimal residual disease.
Beyond KRAS, future treatments will rely on novel vulnerabilities and synergistic combinations. Targeting MTAP loss with PRMT5 and MAT2A inhibitors is a promising strategy, while c-MET targeted therapies are being investigated to overcome resistance. Ultimately, these ongoing advances in precision medicine provide renewed hope for overcoming PDAC.
This presentation will synthesize the latest clinical data on KRAS inhibitors and vaccines, highlighting how these complementary innovations will shape the next generation of precision oncology for PDAC.